Artikel Jurnal Penelitian Gastritis Pdf

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Artikel Jurnal Penelitian Gastritis Pdf

Journal Article of Gastritis Research Pdf

ABSTRACT

Chronic gastritis is a histopathological entity characterized by chronic inflammation of the gastric mucosa. Chronicgastritis tends to damage the gastric mucosa and become sequence atrophy to alter gastric physiology. Pepsinogen (PG) can be used as a biopsy of urticology, Õ as a clinical application to evaluate gastric inflammation. Different cellular origins of PG I and PG II are important because changes in their serum concentration can correlate with some histological gastric anomalies. To determine the correlation between gastritis levels and PG I / II ratio (PGR) in patients with chronic gastritis, we conducted an analytical cross sectional study in 64 consecutive gastritis patients. Mucosal gastric dyspepsia patients who have endoscopic biopsy of uppergastrointestinal 2 in anthrum and 2 in the corpus are examined histologically using the Sydney Updated System (USS) by two pathologically independently, and also serum examined for PG I, PG II, and H. pylori IgG. The degree of gastritis is calculated by the USS method. Pepsinogen examination uses the ELISA method, but H. pylori IgG examination uses immunochromatography (ICT) test methods with local reagents. H. pylori is positive if serologically positive for H. pylori and or histologically. positive pylori. The interobserver agreement for histopathological abnormalities was examined using the kappa test. Differences in PGR and severity of gastritis between subjects with positive H. pylori and negative H. pylori were identified using the Mann-Whitney U test. Correlations between gastritis severity and PGR were identified using the Spearman test and the effect of USS and H. pylori total scores for PGR. identified using dummy regression, also to determine the effect of the total score of USS PGR and H. pylorito identified by using dummy regression. A p value less than 0.05 is considered statistically significant. There were 64 chronic gastritis who averaged 45.9 ± 15.5 years old, consisting of 44 men and 20 women. The levels of PG I218.70 (53.90 Ð 530.00) mg / L, PG II 15.72 (2.84 Ð 59.25) mg / L, and PGR 12.66 (28.97 Ð 5.80) . Interobserver agreement from gastricistologic examation showed moderate to large criteria (k = 0.590 7 0.795) with polymorphonuclear activity k = 0.795, gland atrophy k = 0.591, H. pylori density k = 0.727, chronic inflammation k = 0.629, and intestinal metaplasia k = 0.778 . The frequency of gastric mucosal abnormalities as infected with H. pylori was 28.1%, inflammation was 100.0%, polymorphonuclear activity was 22.8%, atrophy was 37.5%, and intestinal metaplasia was 6.2%. The total score of USS was from 1 to 9 and most had scores of 1 and 2 with a frequency of 17 (26.6%) and 15 (24.4%) respectively. Subjects with H. pyloriinfection had lower PGR than uninfected subjects (11.2 ± 4.3 mg / L vs. 15.0 ± 5.1 mg / L, p = 0.001; Mann-Whitney U test), and also subjects with H. pyloriinfection had higher rates of gastritis than those not infected subjects with both levels of inflammation, polymorphonuclear activity, and atrophy (p = 0,000, p = 0.004, p = 0.041 respectively; Mann-Whitney Utest). There was a significant inverse correlation between USS and PGR total scores (r = -0.470, p <0.0001; Spearman test). Significant effects of total USS scores and H. pylori positivity to PGR (F = 7.015, p = 0.002; dummy regression), but only a significant coefficient of USS total scores (t = -2.030, p = 0.047), but H.pylori positvity does not affect the significance of PGR (t = -1.199, p = 0.235). The total USS score affects PGR by 15.4% (adjusted R2 = 0.154, F = 12.504, p = 0.001; linear regression) with the regression coefficient of -0.933 (t = -3.536, p = 0.001). H. pylori and PGR serology can be used to determine USS total scores significantly (F = 9,498, p <0,0001; dummy regression) and both significant regressions (t = -3,417, p = 0,001; t = 2,360, p = 0.021 each; dummy regression) how can a biopsy be carried out Ô with the formula otal total score of USS = 6,786-0,169.PGRÕ for positive subjects of H. pylori and otal total score of USS = 5,258Ð 0,169.PGRÕ for negative subjects of H. pylori. In conclusion that there was a significant inverse correlation between the total USSscore and PGR, the "biological biopsy" formula for determining the total USS score was "total score of USS = 6,786" 0.169.PGRÕ for H. pyloripositive subjects and "total score USS 5,258" 0,169.PGRÕ for H Subjects negative pylori.

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